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  Discussion on Poco Bueno line and Hyperelastosis Cutis
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Moderator:
DrO

Posted on Thursday, Mar 11, 2004 - 10:58 am:

There has been an increasing incidence of hyperelastosis cutis (HC) (also called hereditary equine regional dermal asthenia (HERDA)) in Quarter Horses. The Poco Bueno line is now believed to be associated with this hereditary condition, see article Equine Diseases » Skin Diseases » Epitheliogenesis Imperfecta (EI) & Hyperelastosis Cutis for more.
Member:
Sully

Posted on Monday, Jan 23, 2006 - 1:43 pm:

Hello Dr. Oglesby,

Not sure if if was mentioned on the site yet, that they have a urine test that can be given to a foal after birth to determine if it is inflicted with HC. This is what Ann Rashmier told me last week. It will not be able to say just how severe the affliction will be.

Respectfully,

Nancy
Moderator:
DrO

Posted on Monday, Jan 23, 2006 - 8:29 pm:

Thanks Nancy,
Not only have I not mentioned it but I have missed this bit of news. Who do you send the urine to and what are they testing for in the urine? I know the UCal folks are working on genetic markers but have not heard on any break throughs.
DrO
Member:
Sully

Posted on Monday, Jan 23, 2006 - 9:44 pm:

Hello Dr O.

She said that you would have your Vet. send it to MSU. If you have the time, you might want to give her a call. I was told that morning's are the best time to reach her. When I told her I had not heard of this test before, she said that they needed to get the information out better.

Nancy
Moderator:
DrO

Posted on Tuesday, Jan 24, 2006 - 7:35 am:

I will check out MSU and see if I can get details.
DrO
Member:
Bucky

Posted on Tuesday, Jan 24, 2006 - 11:49 am:

Do I understand correctly this disease only affects foals?

My mare is a way-off decendent of Poco Bueno, she is 7, is there any reason to worry?

This sounds like a good reason not to breed her, is that true?
Member:
Christel

Posted on Tuesday, Jan 24, 2006 - 12:22 pm:

Mandy, if a horse has it, it will have it as a foal, many times tho the owner doesn't know the horse has it until it starts wearing a saddle, where the saddle will literally make the skin tear.
Horses that are afflicted w/ herda have to come from 2 horses that are carriers. If only one of the parents is a carrier the foal can be a carrier too (25% chance), but will not have the disease.
Hopefully they will have a dna test soon. Even if your mare was to test positive as a carrier, you could still breed her, but you wouldn't want to breed her to another carrier. Your mare could still throw another carrier, and some would say you would be unethical to breed her anyhow.
It will be interesting to see how this pans out, both Doc O lena and Dry Doc (the only foals of Poco Lena) both are confirmed carriers. What both have done for the cutting industry is awesome- it is hard to find a cutting horse w/o these lines and often times they show up 5-6 times in one pedigree.
I have a Poco Pine grand daughter- my best broodmare- and I am almost certain her dam was a carrier, I for one cannot wait for a dna test. I have yet to see any herda in any of my mares foals, but I have never really linebred her.
Hope I helped.
Member:
Bucky

Posted on Tuesday, Jan 24, 2006 - 1:28 pm:

Hi Chirstella,

That was very interesting and it did help. My mare is VERY thin skinned, and very sensitive. I can't curry comb her, she reacts like there are cat claws going down her back. I thought it was just how she was but this kind of puts a new twist on things.
M
Member:
Sully

Posted on Tuesday, Jan 24, 2006 - 1:44 pm:

Mandy....I took this from an article off the internet. Hope it helps!
Under the genetic law of averages, here’s how it works, according to the two researchers. When a normal horse (one that is not a carrier) is crossed with a carrier, 50% of the offspring will be carriers and 50% will be normal. When a carrier is crossed with a carrier, 50% of the offspring will be carriers, 25% will be normal and 25% will be afflicted (can develop the clinical signs of HC). When an afflicted horse is crossed with a carrier, 50% of the offspring will be carriers and 50% will be afflicted. When an afflicted horse is crossed with a normal horse, 100% of the offspring will be carriers.

Nancy
Member:
Christel

Posted on Tuesday, Jan 24, 2006 - 3:20 pm:

Thanks Nancy, I have several articles on herda, my problem is finding them. I was quoting from memory which isn't alway accurate. I like my memory better-lol- I was thinking 25% instead of the 50% you quoted.
Mandy not for sure if your mare being thin skinned is a sign. From what I remember reading, the skin is very loose on back and wither area, can easily be maniuplated. Bet Nancy can give more info, since she is looking at her articles.
Chris- need to change profile- sorry for long name.
Moderator:
DrO

Posted on Wednesday, Jan 25, 2006 - 7:25 am:

Mandy's horse does not have HC. The signs of this disease are far worse than sensitivity to brushing. Nancy's description is correct as HC has a simple recessive pattern of inheritance, for more about all this see the article associated with this discussion.
DrO
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