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This is an archived Horseadvice.com Discussion. The parent article and menus are available on the navigation menu below:
HorseAdvice.com » Diseases of Horses » Lameness » Diseases of the Lower Limb » Suspensory Desmitis, Strain, & Sprain »
  Discussion on Research: How good is ultrasound at detecting suspensory lesions?
Author Message
Moderator:
DrO

Posted on Monday, Jan 4, 2016 - 5:23 pm:

I cannot emphasize enough the importance of excellent equipment in the hands of a experienced ultrasonographer. Given those parameters I believe this is true.
DrO

Equine Vet J. 2015 Dec 29.
Proximal suspensory desmopathy in hindlimbs: a correlative clinical, ultrasonographic, gross post mortem and histological study.
Dyson S1, Murray R1, Pinilla MJ2,3.

Author information:
1Centre for Equine Studies, Animal Health Trust, Lanwades Park, Kentford, Newmarket, Suffolk, CB8 7UU, UK.
2Centre for Preventative Medicine, Animal Health Trust, Lanwades Park, Kentford, Newmarket, Suffolk, CB8 7UU, UK.
3Finn Pathologists, The Veterinary Laboratory, Hoxne Road, Diss, Norfolk, IP21 5TT, UK.

Abstract
REASONS FOR PERFORMING STUDY:
It has been suggested that ultrasonography is unreliable for the detection of hindlimb proximal suspensory desmopathy (PSD) based on comparison between ultrasonographic and magnetic resonance images.

OBJECTIVES:
To compare ultrasonography with gross and histopathological post-mortem examination in horses with PSD diagnosed based on ultrasonography and control horses.

STUDY DESIGN:
Retrospective descriptive study.

METHODS:
Part 1 - Nineteen horses with hindlimb PSD and 10 control horses were humanely destroyed. Twenty control limbs and 37 lame limbs were examined grossly and 40 suspensory ligaments (SLs) were examined histologically and graded blindly. Part 2 - Three horses with recurrent lameness after surgical management of PSD and four with PSD were assessed ultrasonographically and by gross post mortem examination.

RESULTS:
Part 1 - Ultrasonographic lesions were graded moderate in 31/38 (81.6%) and severe in 7/38 (18.4%) lame limbs; in 4/37 (10.8%) limbs adhesion formation between the proximal aspect of the SL and the accessory ligament of the deep digital flexor tendon was predicted. Gross post mortem and histological examinations of control limbs revealed no abnormalities. Gross post mortem examination revealed substantial adhesions between the proximal aspect of the SL and adjacent soft tissues in 10/37 (27.0%) lame limbs; in 10/37 (27.0%) limbs there were adhesions between the body of the SL and the mid plantar aspect of the third metatarsal bone, extending distally in 6 (16.2%) limbs. Histology revealed abnormalities (grades 1-3) of the collagenous tissue in 25/36 (69.4%) limbs; muscle was abnormal (grades 1-3) in 35/36 (97.2%) limbs and adipose tissue (grades 1-3) in 16/36 (44.4%) limbs. Part 2 - Adhesions between the SL and adjacent soft tissues were predicted ultrasonographically and confirmed post mortem.

CONCLUSIONS:
Ultrasonography was reliable for the detection of SL pathology based on histology as the gold standard. Adhesions may be a reason for recurrent lameness after surgery.
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