Navicular Syndrome- lame mare MRI

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    • #17141
      mokuc
      Member

      I have a 22 yr old Stb mare that toes in badly. She came to me 5 years ago and was sound in shoes and pads for light riding. 18 months ago I changed farriers and her toe was allowed to grow very long. She went lame in the left front and has never recovered. Radiographs did not show anything conclusive. A Nerve block improved soundness and coffin joint injections did not help. Katie appears more comfortable with a shorter toe but is lame at the trot in circles and on hard ground.I just got back from having an MRI for her and the issue they pinpointed is extensive moderate desmopathy and mild to moderate enthesopathy affecting the medial collateral ligament of the distal interphalangeal joint. There were other things wrong as well but they believe this is the most relevant. Can you suggest any treatments, hoof care, etc. that would be helpful? I am interested in this mare’s long term comfort and I want to make sure I do the right thing. Thank you. I can send the MRI report but I’m not sure how to attach it.

    • #17156

      Hello mokuc,
      How long has she now been lame? Concerning the desmopathy how much edema, fibrosis, and calcification? Has the coffin joint fluid been evaluated?

      One of the problems with horses that have long established lower limb deformities (“toes in badly”) is knowing what is the best way to balance the foot so that a minimum of stress is placed on the soft tissues that support the foot including the medial collateral ligament of the coffin joint. This balance is not just with respect to the length of the toe but both anterior-posterior balance and medial-lateral balance. Right now it appears that the 3 years of soundness give us a good idea of what was a good trim for this horse. What would it take to have the horse returned to that state?

      If there are some acute treatable causes of lameness they are likely to be due to inflammation secondary to the instabilities that resulted in injury (maybe exacerbated by the trimming but possibly the end stage of what has long been a worsening problem), anti-inflammatory drugs and rest are likely to help. What did the MRI folks have to say with respect to treatments?
      DrO

    • #17157
      mokuc
      Member

      Here is the text of the MRI report. The MRI vet believes that this lameness is the result of a lifetime of crooked legs and balance issues with a fairly large horse. I have tried 2 other farriers and a barefoot farrier. He has kept her toes short and based on her collateral groove depth and my inexperienced visual judgement she appears balanced. She is moving better with this trimmer than the previous 2 others. Here is the report.

      “Katie Girl” has had a low grade LF distal limb lameness for over 18 months. The last time “Katie Girl” underwent a comprehensive lameness evaluation, including diagnostic peripheral nerve anesthesia was in June, 2018. At that time “Katie Girl” was noted to be 2/5 lame LF most noticeable on a circle to the right. PD nerve anesthesia improved the lameness by 75% and she was then noted to be 1-2/5 lame RF on a left circle. PD nerve anesthesia of the RF abolished the lameness.

      “Katie Girl” was not evaluated again for lameness by Genesee Valley Equine Clinic until July 3, 2019. “Katie Girl’s” owner, Maureen, had been away for a few weeks and upon her return felt that the mare was very off in both front feet and expressed some concern about laminitis. An evaluation revealed a baseline level of soundness, but DP and lateral radiographs of both front feet were taken to check for evidence of rotation of the distal phalanx.

      Since this last evaluation, the LF lameness has waxed and waned. “Katie Girl” appears to be more comfortable when her toes are shortened. “Katie Girl” is and has been barefoot. MR imaging requested by Maureen. It was recommended that prior to coming in for advanced diagnostic imaging a comprehensive lameness evaluation be repeated but due to environmental restrictions this was not able to be performed.

      Left fore: There is mild to moderate synovitis and mild arthrosis affecting the distal interphalangeal joint with mild to moderate joint capsule enthesopathy characterized by osseous proliferation on the dorsal aspect of the middle phalanx. The navicular bone has mild distal margin remodeling. Multifocal mild fiber abnormalities are present in the deep digital flexor tendon at the level of the navicular bone. There is focal mild to moderate resorption of the palmar aspect of the medial fossa of the middle phalanx at the attachment of the medial collateral ligament of the distal interphalangeal joint. There are moderate fiber abnormalities and multifocal fiber disruption in the medial collateral ligament of the distal interphalangeal joint through out its length. There is mild to moderate ossification of the collateral cartilages of the distal phalanx. There is mild to moderate remodeling of the medial fossa of the distal phalanx at the insertion of the medial collateral ligament of the distal interphalangeal joint characterized by resorption and enthesophyte formation. There is peripheral study artifact preventing complete evaluation of the dorsal distal aspect of the distal phalanx and the associated lamina, and there are probable laminar and possible distal phalanx defects in this region.

      Right fore: There is mild to moderate synovitis and mild arthrosis affecting the distal interphalangeal joint. There is a laminar and osseous defect on the solar margin of the distal phalanx medial of midline. The navicular bone has mild palmar fluid and mild distal margin remodeling. There is mild bursitis in the proximal recess of the navicular bursa characterized by increased fluid as well as focal lateral synovial proliferation in the distal recess. There are mild to moderate fiber abnormalities in the medial lobe of the deep digital flexor tendon at the level of the middle phalanx with associated lobe enlargement. There is mild to moderate fiber abnormalities in the medial collateral ligament of the distal interphalangeal joint at the distal aspect of the middle phalanx. The appearance of the lateral collateral ligament of the distal interphalangeal joint is consistent with magic angle effect. However, mild

      [Report of Imaging Findings Referring Practice Dr. Stacey Kent VMD EQUIGEN, LLC Thursday, December 5, 2019 MR (Magnetic Resonance) Imaging Details Report created through Asteris Keystone Teleconsultation Page 1 of 2 Dr. Natasha Werpy DACVR DVM Equine Diagnostic Imaging, Inc.]

      degenerative injury cannot be ruled out with this appearance. There is mild to moderate remodeling of the fossae of the distal phalanx at the insertions of the collateral ligaments of the distal interphalangeal joint characterized by resorption and enthesophyte formation. The extensive moderate desmopathy and mild to moderate enthesopathy affecting the medial collateral ligament of the distal interphalangeal joint is the most clinically relevant finding in the left front foot. The right front foot has mild to moderate desmopathy affecting the medial collateral ligament of the distal interphalangeal joint, focal mild to moderate deep digital flexor tendinopathy and mild navicular bone fluid. In addition, there is a solar laminar and distal phalanx defect in the right front foot and probable laminar and possible distal phalanx defects in the left front foot. Further investigation of the front feet is recommended as a space occupying lesion, such as a keratoma, should be considered for these findings. The remaining findings on this study should be clinically correlated. Osseous fluid can be the result of contusion and/or degenerative injury. Osseous fluid as a result of contusion typically resolves with rest, while degenerative injury can result in persistent fluid that may be associated with chronic lameness.

      Conclusions:
      Left fore: Mild to moderate synovitis and mild arthrosis and mild to moderate joint capsule enthesopathy, distal interphalangeal joint Mild distal margin remodeling, navicular bone Multifocal mild deep digital flexor tendinopathy Extensive moderate desmopathy and mild to moderate enthesopathy, medial collateral ligament of the distal interphalangeal joint Mild to moderate collateral cartilage ossification, distal phalanx Peripheral study artifact and probable laminar and possible osseous defects, distal phalanx and the associated lamina
      Right fore: Mild to moderate synovitis and mild arthrosis, distal interphalangeal joint Laminar and osseous defect, distal phalanx solar margin Mild palmar fluid and mild distal margin remodeling, navicular bone Mild navicular bursitis Focal mild to moderate deep digital flexor tendinopathy Mild to moderate desmopathy, medial collateral ligament of the distal interphalangeal joint Probable magic angle effect, lateral collateral ligament of the distal interphalangeal joint Mild to moderate insertional enthesopathy, collateral ligaments of the distal interphalangeal joint

    • #17211

      Considering the wide range of lesions and the several possible chronic causes of lameness that can be inferred from them, my original suggestions stand, more specifically:
      While lame paddock rest, assuming she will be quiet there, and NSAID’s. When sound return to pasture. Remaining sound at pasture light riding might be attempted.

      Steroid injections in the coffin jt (easy) and navicular bursae (difficult) may be helpful. My choice would be methylprednisolone (DepoMedrol) due to its longer action however some veterinarians believe that there are more deleterious effects on the joint and systemically than with other medications.

      A common recommendation would be shock therapy, and this would relieve the lameness. However it should be looked upon as a neurectomy at least early on. Considering the permanent lesions I don’t think the horse should be exercised until the nerve blocking effects wear off.

      Of course all these treatment recommendations have downsides and possible contraindications. You can read more about them as we specific articles on all these, just run a search on each subject. These suggestions need to be discussed, pros and cons with your veterinarian for only you and (s)he can come to a consensus as to what is best for horse.
      DrO

    • #17213
      mokuc
      Member

      Thank you very much! Am I correct in understanding that shock wave will relieve pain but not help with healing the problems that exist? Does it only kill pain or does it have long term beneficial effects for a horse like Katie? The last thing I want to do is make her problems worse for a temporary fix. My vet is out of town and we will speak next week but the more I know about this the better. If this was your long term, family pet, keeper horse- what would you do?

    • #17260

      ECSWT has wide ranging effects on different tissues but especially bone. I was about to refer you to the article on shock wave therapy but instead went to it to see what was there then I used the research button at the bottom of the article (there is one at the bottom of every article labelled PUBMED that returns the last 20 years of research on the articles topic including what was published yesterday) and found there has been little change in the information on ECSWT over the last several years. That which has been published has not been very positive with the exception of wound healing and decreased granulation tissue. There have been negative effects on some normal soft tissue. It still remains to be seen if there may be a positive effect of some types of chronically damaged soft tissue. Read the article and take a look at the research and see what you think. Based on this information I would view the use of ECSWT as experimental (as the article concludes).

      If this were my horse I would be following the recommendations I have given so far as your stated goals would likely be mine also. My expectations would be to make this horse pasture sound and expect anything more than a occasional walk under saddle unlikely. Your goals, and to some degree your resources, determine what you do.
      DrO

    • #17278
      mokuc
      Member

      Thanks very much! I read more about ECSWT as well as IRAP and PRP.There just aren’t any magic fixes:-). I’m going to work with my farrier to understand more about keeping her feet balanced. She gets done every 4 weeks and I’m not sure if anything can be improved. In the spring I am going to redo her paddock so that she has better footing in our muddy, snowy, icy winters.I always want to do something to try and “fix things” but it doesn’t look like this is possible. Katie is a pleasure to have around and retirement from riding her seems to be the right thing.

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